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Indeed, we demonstrated that GSH finely compensates the decline of SOD1 activity in: (i) cells expressing less active SOD1 mutant found in familial amyotrophic lateral sclerosis (Vigilanza et al., 2008)
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Reporting of total pyridine nucleotide levels after tissue lysis is often performed, but can yield data of little biological relevance, especially when working with isolated mitochondrial extracts
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