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However, under conditions of heightened oxidative stress, NO can react with superoxide to form peroxynitrite, thereby exacerbating oxidative damage

Figure 1

Alterations in the normal function of excitatory amino acid carrier 1 affect cysteine transport and GSH synthesis impairing zinc homeostasis (the thiol group of GSH can function as a principal Zn 2+ chelator for the maintenance of Zn 2+ homeostasis in neurons) and oxidative stress, enhancing susceptibility to ischemia-induced neuronal cell death in the hippocampus (162164)

The standard treatment, recombinant tissue-type plasminogen activator (rt-PA), is effective initially, but its efficacy declines rapidly over time as the risk of complications, such as hemorrhagic transformation and blood-brain barrier dysfunction, increases

6c), Salviolone group ( P = 0.0092