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When subjected to enzyme kinetics tests and compared to allopurionol, it was concluded that its inhibitory activity occurs in a mixed way

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This effect was reversed by co-expression of the wild-type PGRMC1 but not of the Y113F mutant, suggesting that PGRMC1 enhances doxorubicin resistance of cancer cells by facilitating its degradation via cytochromes P450

After an 1 h of magnetic stirring at room temperature, P3 (300 mg) was introduced into the reaction system

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