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For example, peak levels of ovarian hormones during proestrus is correlated with increased levels of proliferation in the SGZ [138, 139]

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By activating transcription factors like NF-B and activator protein 1 (AP-1) and upregulating kinases like phosphatidylinositol 3-kinase (PI3K) and mitogen-activated protein kinases (MAPKs), ROS generation can cause pro-inflammatory responses (Chen and Kunsch 2004)