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The proposed mechanism involves maintaining synaptic vesicle integrity, competing for receptor sites, and potentially substituting for depleted neurotransmitters

The quest to slow ageing through drug discovery

doi: 10.1038/s41598-025-92868-y 74 PereiraAGEchaveJJorgeAOSNogueira-MarquesRNur YuksekEBarcielaPet al

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pointed out that renal hypoxia contributes to heightened T cell infiltration and kidney damage, partially mediated by HIF-1-induced metabolic reprogramming in lupus-prone mice (MRL/lpr mice)