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Spondylocheiro dysplastic form of the Ehlers-Danlos syndromean autosomal-recessive entity caused by mutations in the zinc transporter gene SLC39A13

These findings collectively suggest that metastatic C42B augments ROS-regulating enzymes as well as the major antioxidant system, glutathione production, which may dramatically alter neutrophil redox metabolism and ROS in the tumor-bone microenvironment