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Certain biochemical and physiological functions of plant GSTs are characteristic of a given plant-pathogen interaction type (e.g., glucosinolate metabolism, detoxification of mycotoxins), while other functions may be common for several or all interaction types, e.g., the control of plant cell death (oxidative stress) by GSTs and regulation of plant GSTs by various hormones and transcription factors
The protective effect of PPAR agonists against APAP-induced toxicity emphasizes the importance of fatty acid metabolism in this process (Patterson et al
Some of these claims have clinical grounding, while others stretch the evidence considerably
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This dissociation between anti-inflammatory efficacy and bone-preserving capacity suggests that suppression of systemic inflammation may not be sufficient to reverse or prevent skeletal damage, especially in models with advanced joint destruction
