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Apolipoprotein B (ApoB) directly reflects the number of circulating atherogenic lipoprotein particles, while lipoprotein(a) [Lp(a)] represents a genetically determined, independently atherogenic, proinflammatory, and potentially prothrombotic particle

Most evidence is still early-stage or preclinical

Researched for Preclinical (mouse-only) metabolic research as a selective inhibitor of NNMT (nicotinamide N-methyltransferase)

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Its structure gives it several potentially harmful properties: it carries cholesterol, contributes to arterial wall lipid deposition, promotes inflammation, transports oxidized phospholipids, and may influence thrombosis and calcific aortic valve disease