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Our initial attempts to hydrophobize the surface of GSH-AuNCs suggested a ligand exchange reaction to displace the glutathione molecules with a hydrophobic thiolated ligand (e.g

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The transcriptional upregulation of ISR was slightly weaker in the no-Trp + BSO group compared with the no-Cys group, notwithstanding the similar level of p-eIF2 (Fig

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