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CaMK4-dependent activation of AKT/mTOR and CREM- underlies autoimmunity-associated Th17 imbalance

doi: 10.1111/all.13401 3

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Once bilirubin has been formed it can interact with free oxygen radicals, producing its own oxidation which transforms it once again into biliverdin (1, 13).This reconversion is catalyzed by BVR which, by virtue of the same, detoxifies up to 10,000 times the oxidant excesses (1) (figure 3)