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Consistently positive results across these diverse injury models suggest the peptide works through fundamental mechanisms common to all wound healing rather than addressing only specific wound characteristics

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The peptide interacts with the nitric oxide (NO) system, modulating vascular tone and cell signaling at the microenvironmental level to support the transition from the inflammatory phase to the proliferative phase of healing

The mechanism involves both the direct cortisol reduction and the improved sleep quality that supports stress resilience

The fate of taspoglutide, a weekly GLP-1 receptor agonist, versus twice-daily exenatide for type 2 diabetes