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Clin Endocrinol 7:481, 1977

Consequently, simulations suggest that while doxorubicin redox metabolism impacts GSH metabolism, the concentrations required to illicit a toxic response, with either a single or extended exposure, reside outside that of the therapeutic dosing range

Without adequate glutathione reserves, acetaldehyde accumulates in hepatocytes and crosses the blood-brain barrier, where it disrupts neurotransmitter synthesis and causes the cognitive symptoms of hangover

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Complementary mechanisms GSH and TXN systems constitute the two thiol-based antioxidant networks that cooperate to restrain ferroptosis 115