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Postoperative intermittent fasting prevents hippocampal oxidative stress and memory deficits in a rat model of chronic cerebral hypoperfusion

ROS were thought to be primarily produced by the NADPH oxidase (NOX) family in phagocytic cells ( in vitro ( Sun indicated that oxidative stress activates Sirtuin 1 (SIRT1), which deacetylates the forkhead box transcription factor O1 (FOXO1) and traps it in the nucleus, enhancing its transcriptional activity

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The viability of the treated cells was presented as percentage of the viability of the non-treated cells, which was considered 100%