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There are many possible reasons for the lack of an effective therapy for AD including the complexity of the disease process and the resulting inability to identify reliable biomarkers

SAG S-acetyl-glutathione SAG S-acetyl-glutathione a delayed release delivery system designed to protect the molecule from degradation by gastric acids in the stomach and carry the SAG to the small intestine where it can be readily taken up intact by chylomicrons in the gut

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Additionally, TCIs are less potent than high-potency TCS, making them less effective for severe or hyperkeratotic CHE and are predominantly indicated for AD, and not CHE [71, 88, 90]

To improve stability, the peptide includes a tyrosine substitution at the N-terminus