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E., Zhang, C., Pollack, A
The primary aim of this study is to elucidate the molecular mechanisms underlying CP-induced ovarian injury, with a focus on oxidative stress, inflammatory signaling, and apoptotic pathways, focusing on nuclear factor-kappa B (NF-B), nuclear factor erythroid-2-related factor 2/heme oxygenase-1 (Nrf2/HO-1), Toll-like receptor 4 (TLR4), nucleotide-binding oligomerization domain-like receptor family pyrin domain-containing 3 (NLRP3) inflammasome, silent information regulator-1 (SIRT1), and other signaling pathways involved in the pathogenesis of ovarian injury caused by CP
Because your body naturally uses and depletes glutathione through daily activities and environmental exposure, a consistent daily dose helps maintain steady levels for ongoing antioxidant support
Brussels, BE
doi: 10.3389/fmicb.2016.01925 Received 22 July 2016 Accepted 16 November 2016 Published 06 December 2016 Volume 7 - 2016 Edited by Miguel Cacho Teixeira, University of Lisbon, Portugal Reviewed by Hans Uwe Dahms, Kaohsiung Medical University, Taiwan
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