For the AOD-9604 deep dive, see What Is AOD-9604
Both promote angiogenesis, but through different upstream pathways (VEGF-mediated for BPC-157, endothelial migration-mediated for TB-500), potentially producing additive vascularisation effects
These compounds have the potential for use in analyzing B 12 pathways or even attacking B 12 -dependent pathogens
Failure to diagnose and treat in time, however, may result in permanent neurological damage, excessive fatigue, depression, memory loss, and other complications
By preventing G-actin from polymerizing into F-actin, TB-500 maintains a pool of unpolymerized actin that allows cells, such as keratinocytes and fibroblasts, to physically "crawl" to the site of an injury