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Alternatively, interventions based on metabolic-epigenetic coupling, such as targeting DGAT2 to modulate lipid droplet-mitochondrial interactions or enhancing metabolic and chromatin states through the NAD + /SIRT pathway, may offer additional avenues

Baxter PS, Bell KF, Hasel P, Kaindl AM, Fricker M, Thomson D, et al

However, obesity-induced chronic inflammation and OS limit treatment efficacy

S -propenyl cysteine, a derivative, can inhibit the NF-B pathway and activate the Keap1/Nrf2 pathway, mitigating inflammation and oxidative stress, thereby supporting cysteine's role in managing autoimmune disorders (Mo et al

Smoking and liver disease