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No phase 2 or 3 human trial has established an optimal oral dose

You want appropriate inflammation that resolves on time

3c and Supplementary Fig

FOXO4-DRI acts as a competitive inhibitor of this interaction binding directly to the transactivation domain 2 (TAD2) of p53, displacing FOXO4, and allowing phosphorylated p53 to be exported from the nucleus and directed to mitochondria, where it activates BAX, triggers caspase-3 cleavage (via the p53/BCL-2/Caspase-3 signalling pathway), and drives cell-intrinsic apoptosis

Over time, there has been a growing interest in understanding the mechanisms and risk factors associated with VAN and discovering ways to mitigate side-effects