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Bernabei, I., So, A., Busso, N

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doi: 10.1097/ALN.0b013e318289bc9b 10 BourneJ.HarrisK

The effect of cortistatin on a wide panel of inflammatory mediators could suppose a clear advantage versus several therapies designed to neutralize single inflammatory cytokines/chemokines or to inhibit intracellular factors involved in inflammatory signaling (i.e., NFkB) that have proven their efficacy in models of neuropathic pain [48,65,66]