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Closed both skin and duodenal defect in BPC 157 rats (fistula closed, b ) (Skorjanec et al., 2015)

Under normal physiological conditions the NO increase might be controlled by S-nitrosylation/glutathionylation of eNOS as an immediate response or by similar modification/activation of SERCA in steady-state regulation

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Furthermore, the review highlights delivery innovations, safety considerations, and translational strategies, thereby providing a theoretical foundation for future research and clinical application of flavonoid-based therapies in AD

Glutathione has also been reported to be a substrate for glutathione peroxidase (GPx) an enzyme which reduces hydrogen peroxide (H 2 O 2 ) in the presence of superoxide dismutase [4]