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This is likely due to increased production of O 2 by the mitochondria, leading to oxidation and inactivation of the ACO-2 substrate, and hence further accumulation of citrate to promote increased mtDNA synthesis

However, it is important to note that the mean birth weights reported in these studies fell within the normal range, and correlation does not necessarily equal causation

But there is more negative news: the targeted therapies that benefit many never-smokers with lung cancer eventually stop working because the tumours develop drug resistance

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Archived from the original on January 23, 2021