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phosphatidylinositols (PIs), ether-linked phosphatidylethanolamines (PEs)] were reduced compared to normal subjects [44]

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Mechanistically, ferroptosis is driven by the accumulation of lethal phospholipid hydroperoxides (PL-OOH), primarily derived from polyunsaturated fatty acid (PUFA)-containing phospholipids

The results may also reflect glutathione breakdown products rather than direct enhancement of the transsulfuration pathway