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NO normally functions to reduce plasma LDL concentration via S-nitrosylation of the LDL receptor adaptor protein ARH, thereby enhancing LDL receptor-mediated clearance of LDL 33

The rational design of these nanocarriers requires synergistic optimization of multiple parameters, including size/morphology (influencing biodistribution and the EPR effect), surface chemistry (affecting stability and biocompatibility), and targeting ligands (enabling cell-specific delivery), collectively aiming to achieve potent and localized ICD induction for enhanced immunotherapy (Yuan F

Springer, 128-135, 2003

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