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While previous research has firmly established its capacity to ameliorate hepatic steatosis, likely by activating the hepatic AMPK signaling pathway to promote fatty acid oxidation and inhibit lipogenesis, and exerting anti-inflammatory effects by suppressing cytokines such as TNF- and IL-6 16 , the present study provides a more unified mechanistic explanation for this

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The common challenges faced in clinical translation include immune rejection, the variability in patient responses, and the need for standardized cell products and delivery protocols

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