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Tumor-associated macrophages can secrete transforming growth factor beta (TGF-) to turn on hepatic leukemia factor (HLF), which enhances the transcription of gamma-glutamyltransferase 1 (GGT1) and promotes resistance to ferroptosis in TNBC [16]

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Direct cellular damage: Lipase directly targets pancreatic cells, disrupting cell membranes and increasing cellular permeability

Clinically, plasma malondialdehyde (MDA)a byproduct of lipid peroxidationserving as a biomarker of oxidative stress severity, with elevated levels correlating with accelerated lung function decline in COPD patients

Although peak plasma ammonium levels are typically in the range of 100500 mol/L in organic acidemia and congenital lactic acidosis, in neonatal onset urea cycle disorders they are generally above 500 mol/L