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Therefore, directing interventions toward iron metabolic pathways and modulating cellular iron homeostasis could present a promising avenue for breast cancer treatment, by utilizing strategies such as iron chelation and addressing iron overload

In addition, classical ferroptosis activators (such as ML-210) or inhibitors (such as ferrostatin-1, Fer-1) modulate intracellular autophagic flux in testicular injury

When these pro-inflammatory mediators reach the hypothalamus, they increased the levels of oxidative stress and neuronal inflammation in the hypothalamus through processes such as microglia proliferation (124, 125)

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