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Overexpression of NLGN2 in adult mice leads to increased localization of GABAergic synaptic proteins at presynaptic and postsynaptic sites, associated with deficits in hippocampus-dependent behaviors and memory formation (84)

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In the hours to days following the initial injury, neuronal and glial dysfunction, metabolic changes, neuroinflammation, and the release of signaling and inflammatory molecules from neurons, glial cells, and immune cells occur