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Nrf2 activation regulates antioxidant responses to modify cellular redox states from predominantly pro-oxidant to antioxidant, and, in an antioxidant environment, macrophage phenotypes shift from M1 pro-inflammatory to M2 anti-inflammatory, reducing the probability of cytokine storms, ARDS, and lethality (Tan et al., 2016

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Tissue Remodeling: Promotes the development of Type IIa oxidative muscle fibers, which are highly resistant to fatigue

Instead of blocking pain signals downstream, certain peptides appear to promote actual nerve fiber regrowth, reduce the neuroinflammation driving ongoing damage, and restore function rather than merely masking dysfunction

Ventura, M., et al