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Interestingly, much of the research has been based on doses far exceeding the recommended daily intake, both in individuals with pathological conditions and in healthy subjects

Activation of the PI3K/AKT/mTOR pathway is commonly observed in TNBC, often due to gain-of-function mutations in PI3KCA and loss of function in PTEN (Tian et al., 2022)

Such patients often are prescribed supplemental B

among them, splice variant 1 (SV1) exhibits the greatest similarity to GHRH-R and remains functional upon GHRH stimulation, eliciting cAMP signaling and mitogenic activity (97)

Authors contributions LT, WW, WZ and KC performed the experiments