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Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1
Super GHK-Copper Cream: Water, ethylhexyl palmitate, squalane, stearic acid, cetyl alcohol, glyceryl stearate, PEG-100 stearate, PPG 2 myristyl ether propionate, copper tripeptide-1 (GHK-Copper), phenoxyethanol, C10-30 cholesterol/lanosterol esters, polysorbate 80, allantoin, leucine, lavandula angustifolia (lavender) oil, aloe barbadensis (aloe vera) powder, sodium salicylate, vitamin E TPGS (tocophersolan), vitamin E (as d-alpha tocopherol plus mixed tocopherols: d-beta, d-delta and d-gamma tocopherol)
The same 30 mg vial reconstituted with 1.5 mL creates a 20 mg/mL concentration
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