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The limited efficacy of even potent immunosuppression in the later stages (Wolinsky et al., 2020) is likely due to a waning of the acute/adaptive auto-immune component, rather than restricted access of biologicals to the CNS (as an example of adequate access of biologicals to the brain, systemic administration of anti-amyloid antibodies in Alzheimers has now been shown to be effective at reducing amyloid load (Jucker and Walker, 2023

It is important to understand that BPC 157 is still considered experimental, and most research has been conducted in animal studies rather than large human clinical trials

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Consequently, these findings raise important questions regarding the extent to which the effects of GLP-1 receptor agonism and amylin receptor activation may be additive or synergistic across different metabolic phenotypes