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No significant adverse effects have been reported from animal studies or first-hand human experiences

While ubiquinone is a co-enzyme required for the function of oxidative phosphorylation, it is not considered a vitamin as it is synthesized from other amino acids

The impressive data supporting the role of deacetylation by HDACs in epilepsy arises from the study that the interaction of HDAC3 with MADS-box domain of MEF2 results in MEF2 deacetylation in nucleus and the decrease of MEF2 transcription [74]