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Nrf2 activated by Sal bind and upregulated the antioxidant response element such as heme oxygenase-1 (HO-1), SOD, and GSHPx [368]

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They reported that both HGF and c-Met were up-regulated in the hyperproliferative epithelium during wound repair in mice, suggesting that HGF and c-Met signal acted in an autocrine manner to promote wound healing