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One could speculate that a key treatment strategy for progressive MS could involve targeting CNS-resident B cells, not aiming to suppress their traditional immunological (mis)behavior, but instead to quell a yet-to-be-discovered unconventional action, possibly related to secretion of an MS-specific myelin toxin into the CSF

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TLR2, a key member of the toll-like receptor family, is recognized for its ability to bind various natural ligands and can participate its involvement in the progression of diverse diseases [35, 36]

95% CI 1.473.63) [96]

Moreover, advances in formulation technologies, such as encapsulation and mitochondria-targeted delivery systems, should be directly tested in clinical trials to determine whether improved bioavailability enhances therapeutic efficacy