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N-Acetyl-L-cysteine ethyl ester has the potential to substitute NAC as a mucolytic agent, and as a GSH-related antioxidant [1]

Unlike BPC-157, which is a synthetic fragment with no natural full-length counterpart, TB-500 mirrors an endogenous peptide with well-characterized biological roles including actin sequestration, cell migration promotion, and tissue repair coordination

Interestingly, estrogens, especially 17-estradiol, have been recently shown to favor a resistance towards ferroptotic death through both genomic and non-genomic mechanisms [40]

But surrounding this core is the ischemic penumbra, a larger region of at-risk tissue that is damaged but not yet dead

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