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showed that the activation of ferroptosis by erastin increased the levels of lysosome-associated membrane protein 2a (LAMP2A) to promote CMA, which, in turn, triggered the degradation of GPX4

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MIA reprograms microglia in a phenomenon known as trained immunity (otherwise known as innate immune memory) to enhance immune responses or suppress immune pathways to confer immune tolerance [51] via epigenetic modulations involving histone modifications, DNA methylation, chromatin remodeling, and miRNAs [52]