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Intraperitoneal administration of 3DNA-si.IDH1 significantly slowed xenograft growth in mice, as compared to a variety of control arms (Fig

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Data availability A dataset with deidentified participant data and a data dictionary will be made available upon reasonable request from any qualified investigator, subject to a signed data access agreement

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This pathway activation enables: Accelerated migration of repair cells to injury sites Improved cell survival under oxidative stress conditions Enhanced F-actin formation essential for cellular movement Increased fibroblast outgrowth and spreading capacity Research using FITC-phalloidin staining demonstrated that BPC-157 potently stimulates F-actin formation, working synergistically with TB-500s actin-sequestering functions to provide comprehensive cytoskeletal support for cellular mechanics throughout the healing process