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By contrast, the effects of anti-cancer agents that seek to induce ferroptosis via GSH depletion (e.g., cyst(e)inase, [Cramer et al, 2017]) could be blunted if combined with chemotherapeutics such as gemcitabine and HU, which inhibit the consumption of GSH by nucleotide metabolic processes and also slow DNA replication by blocking elongation (Heinemann et al, 1990

E., Bachtell, R

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HOMA2-IR returns in proportion to regain (about +0.6% at under-25% regain to +39.5% at 50-75%)