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GHK-Cu also modulates gene expressionupregulating genes associated with tissue repair while downregulating inflammatory and fibrotic pathways

Anchor logic: why the two are stacked Think of tissue repair as a construction project with two independent bottlenecks

in these cultured cells cotreatment of klotho and bFGF further increased ERK1/2 phosphorylation, while inhibition of ERK1/2 activation leads to abolition of klotho-induced GH release in normal pituitaries (102)

Animals The study was conducted with male Albino Wistar rats, bodyweight 200 g, 13 weeks old, in-house bred at the animal facility of the Department of Pharmacology, School of Medicine, Zagreb, Croatia