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Since GSH is the final product of the homocysteine metabolism in the transsulfuration pathway by transferring sulfur from homocysteine to cysteine, a deficiency in transsulfuration pathway leads to excessive homocysteine production (hyperhomocysteinemia) and reduced GSH synthesis (159, 160)

When mitochondrial glutathione is depleted, energy production becomes less efficient and mitochondrial DNA accumulates mutations a process associated with aging and age-related diseases

The cystine/cysteine cycle: a redox cycle regulating susceptibility versus resistance to cell death

Fane, M

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