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Hence, ferroptosis is likely not a major contributing factor in APAP-induced hepatotoxicity under physiological conditions

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Competitive cellular uptake of liposomes in the two cell types was visualized using an inverted fluorescence microscope, and fluorescence intensity was semi-quantitatively analyzed with ImageJ software

These observations create a plausible mechanistic pathway in PCOS: metabolic and hormonal perturbations (hyperandrogenism, insulin resistance) promote platelet hyperreactivity and PMP release, which in turn primes neutrophils to NETose within adipose, ovarian, and vascular beds