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Mechanistically, STING was shown to directly interact with YY1, inhibiting its nuclear translocation and consequently promoting transcription of lipocalin-2 (LCN2)

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Across biochemical assays, cell culture models, and preclinical animal studies, mechanistic investigations have focused on how BPC-157 interacts with nitric oxide signaling, angiogenic regulation, cytoskeletal remodeling, and epithelial or connective tissue repair processes

AQP4 on astrocytic end-feet relocates towards the soma, causing astrocyte swelling, capillary compression, and decreased CBF [95,96,97]