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This results from a tyramine-induced depletion of norepinephrine normally present in the readily releasable vesicle pool, and replacement of vesicular norepinephrine with tyramine (which functions as a false- or pseudo- neurotransmitter), which when released has no effect on postsynaptic adrenergic receptors (Figure 1)

For instance, insoluble dietary fibres fermented to acetate (for appetite regulation) (Silva, Bernardi, & Frozza, 2020), butyrate (for memory, mood, cognition) and propionate (preventing diabetes mellitus, obesity and stress) short-chain fatty acids (SCFAs) (Silva et al., 2020)

How many layers and levels are at play