Well, okay, so now let's talk about the agency
Your body produces many peptides naturally, but their levels decline with age
The confocal fluorescence microscopy analysis showed that the three GPI type tags, GST-EGFP-GPI DAF , GST-EGFP-GPI BY55, and GST-EGFP-GPI CEAM7, were efficiently expressed and transported to the cell membrane (Figure 2A)
This could prevent cell death, meaning that glutathione could conceivably operate as a neuroprotective agent one that could slow or stop the progression of PD
Three different tests: Tendon outgrowth , how many cells grow out from a piece of tendon in the dish Wound healing assay , cells are grown as a monolayer, you make a scratch in it and observe how quickly it closes Cell survival , you add peroxide (oxidative stress, which normally kills cells) and observe how many survive What they found: Tenocytes grew faster and in a dose-dependent curve (the more BPC-157, the more growth) The scratch closed 1.8 faster at 1 g/ml BPC-157 Under oxidative stress, 22 % of cells survived without BPC-157, but 67 % with BPC-157 When they blocked the FAK enzyme, the effect disappeared mechanism confirmed Why it matters: This is not just a systemic effect via blood, BPC-157 directly modifies the biology of an individual cell