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This process leads to caspase-1 activation, which facilitates the maturation and release of IL-18 and IL-1

This temporal dissociation suggests that early microglial activation, acting in a neuroprotective fashion, may serve a compensatory or protective role, potentially delaying the onset of clinical symptoms [468]

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Metabolic bone disease in children with transfusion-dependent thalassemia

nor were found susceptible to extraneous stressors, highlighting that Sp exposure did not compromise organismal response to electrophiles (Fig