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Therefore, these results suggest that a low level of PS exposure promotes axonal fusion, while a high level of PS exposure leads to axonal debris

Its unique mechanism of action does not interfere with hormone-related peptides or tissue repair compounds

Molecular mechanisms and pathological consequences of endotoxin tolerance and priming

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In this case molecules are absorbed in the absence of receptors, but rather through charged interaction between the ligand and the glycocalyx of endothelial cells and can be referred to as absorptive-mediated transcytosis (Fig