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Similar results were reported in STEP-HFpEF DM, which included patients with diabetes excluded in STEP-HFpEF [30]

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Because of this, the analogue produces steady, prolonged elevations in GH and IGF-1 instead of the usual short-lived, pulsatile GH spikes

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The current evidence base points to several overlapping molecular pathways operating in parallel: VEGFR2/Akt/eNOS Angiogenic Signalling A primary mechanistic axis involves upregulation of vascular endothelial growth factor receptor 2 (VEGFR2), leading to activation of the Akt/eNOS (endothelial nitric oxide synthase) downstream pathway