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The immune system, designed to protect the body from infection and disease, turns inward

The proposed mechanism of action in vitiligo is by modulation of immune response by reduction of CD4+ and CD8+ T lymphocytic cells, as well as reduction of macrophages

This sensitization to TGF-beta signaling augments expression of matrix genes including collagens, fibronectin, and proteoglycans, while appropriate regulation prevents excessive TGF-beta activity associated with pathological fibrosis

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Frequently Asked Questions What is the difference between acute and chronic inflammation