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In an endothelium-independent way, CGRP, due to its receptors, causes adenylate cyclase activation and cAMP increase, which activates protein kinase A (PKA), and this, in turn, phosphorylates ATP-dependent K+ channels, which leads to Ca 2+ sequestration mechanisms and causes smooth muscle relaxation

In this review, we highlight the epidemiological evidence linking G6PD-d to ASCVD and discuss the literature that supports mechanisms that may explain this link, with a focus on the potential impact of G6PD-d on the immune cells involved in atherosclerosis

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Miyhab KB et al