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Its primary mechanisms include promoting angiogenesis (new blood vessel formation) through VEGF upregulation, modulating the nitric oxide system in a context-dependent manner, activating the FAK-paxillin pathway to drive cell migration and tissue rebuilding, and regulating multiple growth factors (PDGF, TGF-beta, FGF) involved in organized tissue repair

"Thymosin beta4 accelerates wound healing." Journal of Investigative Dermatology , 113(3), 364368

Photograph affected areas in consistent lighting

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Products containing copper salts instead of the peptide deliver unbound copper directly to the skin, which generates reactive oxygen species and can damage the dermal matrix over repeated use